{"id":18842,"date":"2026-05-04T06:19:40","date_gmt":"2026-05-04T06:19:40","guid":{"rendered":"https:\/\/www.diginerve.com\/blogs\/?p=18842"},"modified":"2026-05-12T07:18:53","modified_gmt":"2026-05-12T07:18:53","slug":"important-physiology-questions-for-neet-pg","status":"publish","type":"post","link":"https:\/\/www.diginerve.com\/blogs\/important-physiology-questions-for-neet-pg\/","title":{"rendered":"Important Physiology Questions for NEET PG 2026"},"content":{"rendered":"

Physiology is one of the most important subjects for NEET PG because it forms the conceptual base for Medicine, Pathology, Pharmacology, and many clinical subjects. In NEET PG, physiology questions are usually concept-based rather than memorisation-based, and they often test mechanisms, applied clinical scenarios, graphs, ECGs, spirometry, neuromuscular physiology, renal function, endocrinology, and cardiovascular regulation. The following 20 important questions are prepared in a NEET PG-oriented format to help students quickly revise high-yield topics and understand the core concepts required to solve clinical and integrated MCQs.<\/span><\/p>\n

Here are the Physiology Questions for NEET PG 2026<\/strong><\/h2>\n

Q1. The most important determinant of resting membrane potential in neurons is:<\/b><\/p>\n

A. Sodium permeability<\/span>
\n<\/span>B. Potassium permeability<\/span>
\n<\/span>C. Calcium permeability<\/span>
\n<\/span>D. Chloride permeability<\/span><\/p>\n

Answer: B. Potassium permeability<\/b>
\n<\/span>At rest, the cell membrane is most permeable to K<\/span>\u207a<\/span>, so the resting membrane potential lies close to the K<\/span>\u207a<\/span> equilibrium potential.<\/span><\/p>\n

 <\/p>\n

Q2. The upstroke of the action potential in a neuron is due to:<\/b><\/p>\n

A. K<\/span>\u207a<\/span> efflux<\/span>
\n<\/span>B. Na<\/span>\u207a<\/span> influx<\/span>
\n<\/span>C. Ca\u00b2<\/span>\u207a<\/span> efflux<\/span>
\n<\/span>D. Cl<\/span>\u207b<\/span> influx<\/span><\/p>\n

Answer: B. Na<\/b>\u207a<\/b> influx<\/b>
\n<\/span>The opening of voltage-gated sodium channels causes rapid depolarisation.<\/span><\/p>\n

 <\/p>\n

Q3. A patient has myasthenia gravis. The defect is at:<\/b><\/p>\n

A. Presynaptic Ca\u00b2<\/span>\u207a<\/span> channel<\/span>
\n<\/span>B. Acetylcholine receptor at the neuromuscular junction<\/span>
\n<\/span>C. Acetylcholinesterase enzyme excess<\/span>
\n<\/span>D. Muscle ryanodine receptor<\/span><\/p>\n

Answer: B. Acetylcholine receptor at the neuromuscular junction<\/b>
\n<\/span>Myasthenia gravis is due to antibodies against postsynaptic nicotinic acetylcholine receptors.<\/span><\/p>\n

 <\/p>\n

Q4. The PR interval in ECG represents:<\/b><\/p>\n

A. Ventricular depolarisation<\/span>
\n<\/span>B. Atrial repolarisation<\/span>
\n<\/span>C. AV nodal conduction time<\/span>
\n<\/span>D. Ventricular repolarisation<\/span><\/p>\n

Answer: C. AV nodal conduction time<\/b>
\n<\/span>PR interval reflects conduction from the atria through the AV node to the ventricles.<\/span><\/p>\n

 <\/p>\n

Q5. The second heart sound is produced mainly by:<\/b><\/p>\n

A. Closure of AV valves<\/span>
\n<\/span>B. Opening of semilunar valves<\/span>
\n<\/span>C. Closure of semilunar valves<\/span>
\n<\/span>D. Rapid ventricular filling<\/span><\/p>\n

Answer: C. Closure of semilunar valves<\/b>
\n<\/span>S2 is due to the closure of the aortic and pulmonary valves.<\/span><\/p>\n

 <\/p>\n

Q6. Increased preload causes increased stroke volume due to:<\/b><\/p>\n

A. Bainbridge reflex<\/span>
\n<\/span>B. Frank-Starling mechanism<\/span>
\n<\/span>C. Baroreceptor reflex<\/span>
\n<\/span>D. Cushing reflex<\/span><\/p>\n

Answer: B. Frank-Starling mechanism<\/b>
\n<\/span>Increased venous return stretches cardiac muscle fibres and increases the force of contraction.<\/span><\/p>\n

 <\/p>\n

Q7. In aortic stenosis, the pulse is classically:<\/b><\/p>\n

A. Pulsus paradoxus<\/span>
\n<\/span>B. Pulsus bisferiens<\/span>
\n<\/span>C. Pulsus parvus et tardus<\/span>
\n<\/span>D. Water-hammer pulse<\/span><\/p>\n

Answer: C. Pulsus parvus et tardus<\/b>
\n<\/span>Aortic stenosis produces a slow-rising, low-volume pulse.<\/span><\/p>\n

 <\/p>\n

Q8. The best substance for measuring GFR is:<\/b><\/p>\n

A. PAH<\/span>
\n<\/span>B. Inulin<\/span>
\n<\/span>C. Urea<\/span>
\n<\/span>D. Creatinine<\/span><\/p>\n

Answer: B. Inulin<\/b>
\n<\/span>Inulin is freely filtered, not secreted, not reabsorbed, and not metabolised.<\/span><\/p>\n

 <\/p>\n

Q9. PAH clearance is used to estimate:<\/b><\/p>\n

A. GFR<\/span>
\n<\/span>B. Renal plasma flow<\/span>
\n<\/span>C. Renal blood flow directly<\/span>
\n<\/span>D. Tubular reabsorption rate<\/span><\/p>\n

Answer: B. Renal plasma flow<\/b>
\n<\/span>PAH is filtered and secreted, so its clearance approximates effective renal plasma flow.<\/span><\/p>\n

 <\/p>\n

Q10. Counter-current multiplication in the kidney mainly occurs in:<\/b><\/p>\n

A. Proximal convoluted tubule<\/span>
\n<\/span>B. Loop of Henle<\/span>
\n<\/span>C. Distal convoluted tubule<\/span>
\n<\/span>D. Collecting duct only<\/span><\/p>\n

Answer: B. Loop of Henle<\/b>
\n<\/span>The thick ascending limb actively reabsorbs NaCl and is impermeable to water, creating the medullary gradient.<\/span><\/p>\n

 <\/p>\n

Q11. ADH increases water reabsorption by inserting:<\/b><\/p>\n

A. Aquaporin-1 in PCT<\/span>
\n<\/span>B. Aquaporin-2 in the collecting duct<\/span>
\n<\/span>C. ENaC in the loop of Henle<\/span>
\n<\/span>D. Na<\/span>\u207a<\/span>\/K<\/span>\u207a<\/span> ATPase in the glomerulus<\/span><\/p>\n

Answer: B. Aquaporin-2 in collecting duct<\/b>
\n<\/span>ADH acts on V2 receptors and inserts aquaporin-2 channels in the collecting duct.<\/span><\/p>\n

 <\/p>\n

Q12. In obstructive lung disease, spirometry shows:<\/b><\/p>\n

A. Normal FEV1\/FVC<\/span>
\n<\/span>B. Increased FEV1\/FVC<\/span>
\n<\/span>C. Decreased FEV1\/FVC<\/span>
\n<\/span>D. Increased vital capacity<\/span><\/p>\n

Answer: C. Decreased FEV1\/FVC<\/b>
\n<\/span>FEV1 falls more than FVC in obstructive diseases like asthma and COPD.<\/span><\/p>\n

 <\/p>\n

Q13. Surfactant is secreted by:<\/b><\/p>\n

A. Type I pneumocytes<\/span>
\n<\/span>B. Type II pneumocytes<\/span>
\n<\/span>C. Alveolar macrophages<\/span>
\n<\/span>D. Club cells<\/span><\/p>\n

Answer: B. Type II pneumocytes<\/b>
\n<\/span>Surfactant reduces alveolar surface tension and prevents collapse.<\/span><\/p>\n

 <\/p>\n

Q14. Pulmonary embolism causes:<\/b><\/p>\n

A. Physiological shunt<\/span>
\n<\/span>B. Increased dead space ventilation<\/span>
\n<\/span>C. Reduced anatomical dead space<\/span>
\n<\/span>D. Decreased V\/Q ratio<\/span><\/p>\n

Answer: B. Increased dead space ventilation<\/b>
\n<\/span>Ventilation is present, but perfusion is reduced, so the V\/Q ratio increases.<\/span><\/p>\n

 <\/p>\n

Q15. A right shift of the oxygen dissociation curve is caused by:<\/b><\/p>\n

A. Decreased temperature<\/span>
\n<\/span>B. Decreased PCO\u2082<\/span>
\n<\/span>C. Increased pH<\/span>
\n<\/span>D. Increased 2,3-BPG<\/span><\/p>\n

Answer: D. Increased 2,3-BPG<\/b>
\n<\/span>Right shift means decreased Hb affinity for oxygen and increased tissue oxygen unloading.<\/span><\/p>\n

 <\/p>\n

Q16. Bitemporal hemianopia is caused by a lesion of:<\/b><\/p>\n

A. Optic nerve<\/span>
\n<\/span>B. Optic chiasma<\/span>
\n<\/span>C. Optic tract<\/span>
\n<\/span>D. Visual cortex<\/span><\/p>\n

Answer: B. Optic chiasma<\/b>
\n<\/span>Compression of crossing nasal retinal fibres causes loss of temporal visual fields.<\/span><\/p>\n

 <\/p>\n

Q17. Pain and temperature fibres decussate in:<\/b><\/p>\n

A. Dorsal column<\/span>
\n<\/span>B. Medulla<\/span>
\n<\/span>C. Anterior white commissure of the spinal cord<\/span>
\n<\/span>D. Internal capsule<\/span><\/p>\n

Answer: C. Anterior white commissure of the spinal cord<\/b>
\n<\/span>Spinothalamic tract fibres cross within 1\u20132 spinal segments after entry.<\/span><\/p>\n

 <\/p>\n

Q18. QAldosterone secretion is mainly stimulated by:<\/b><\/p>\n

A. ACTH only<\/span>
\n<\/span>B. Angiotensin II and hyperkalemia<\/span>
\n<\/span>C. TSH<\/span>
\n<\/span>D. ANP<\/span><\/p>\n

Answer: B. Angiotensin II and hyperkalemia<\/b>
\n<\/span>Zona glomerulosa secretes aldosterone in response to angiotensin II and increased serum K<\/span>\u207a<\/span>.<\/span><\/p>\n

 <\/p>\n

Q19. Secretin mainly stimulates:<\/b><\/p>\n

A. Gastric acid secretion<\/span>
\n<\/span>B. Pancreatic bicarbonate secretion<\/span>
\n<\/span>C. Gallbladder contraction<\/span>
\n<\/span>D. Pepsinogen secretion<\/span><\/p>\n

Answer: B. Pancreatic bicarbonate secretion<\/b>
\n<\/span>Secretin is released from duodenal S cells in response to acid and promotes bicarbonate-rich pancreatic secretion.<\/span><\/p>\n

 <\/p>\n

Q20. Ovulation is triggered by:<\/b><\/p>\n

A. FSH surge<\/span>
\n<\/span>B. LH surge<\/span>
\n<\/span>C. Progesterone withdrawal<\/span>
\n<\/span>D. Prolactin surge<\/span><\/p>\n

Answer: B. LH surge<\/b>
\n<\/span>Mid-cycle LH surge causes ovulation and luteinization of the ruptured follicle.<\/span><\/p>\n

 <\/p>\n","protected":false},"excerpt":{"rendered":"

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